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tissue damage to the brain. When this happens suddenly, it is called a stroke. Hypertension, an accumulation of fatty deposits in the arteries, and the interaction with other illnesses that damage the brain can create vascular dementia. Cognitive and psychological changes often accompany the physical brain changes that occur. The nature of these cognitive and psychological changes depends on both the nature of the patient s premorbid level of functioning and the specific areas of the brain that are affected (Desmond & Tatemichi, 1998). Risk of stroke and other vascular dementia increases with age. Parkinson s disease is a degenerative brain disorder that is due to the brain s inability to adequately produce the neurotransmitter, dopamine. The disease includes symptoms such as involuntary tremors and muscle rigidity. About half of Parkinson s disease patients develop dementia during the advanced stages of the illness. Although Parkinson s disease has received a great deal of attention recently due to the illness of the well-known actor, Michael J. Fox, it is a disease more often seen among the elderly. Geropsychologists may be involved with neuropsychological testing to help assess the degree of cognitive and psychological damage as well as assess the strengths remaining in those who experience dementia from vascular disease, Parkinson s disease, or other reasons. These professionals might also participate in individual, family, and group psychotherapy and psychoeducational activities to help both the patient and loved ones cope with the dementia in particular and the general illness. For example, behavior therapy is often used to help patients deal with emotional volatility (Dunkin & Anderson-Hanley, 1998). Cross-cultural research from a variety of different countries and cultures suggest that people with less education are more likely to be diagnosed with dementia than those with higher educational achievements (e.g.,

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The number of helpful or fun activities you can do with a PC is simply staggering: paying bills over the Internet; printing digital photos; scanning old pictures to create long-lasting archival copies; editing home movies, listening to MP3 music les; and so much more. If you are interested in using your computer for more than playing Solitaire, this section of the book will show you numerous ways to make your high-tech experience more rewarding.

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of the neural crest, some SA cells arrive in the adrenal anlagen around embryonic day 105 and are arrested there both developmentally and functionally (a process termed neoteny), while other SA cells continue to migrate and differentiate eventually into sympathetic neurons [Anderson and Axel, 1986; Anderson, 1989, 1993] In the adrenal anlagen, these SA cells differentiate into two distinct catecholaminergic chromaf n cell types, which are characterized as either noradrenergic or adrenergic, that is, each of these related, yet distinct, cell types synthesizes and secretes either noradrenaline (norepinephrine, NE) or adrenaline (epinephrine, E) [Moro et al, 1990; Weiss et al, 1996] Recent studies indicate that the expression of noradrenaline in the NE cells during differentiation is independent of the presence of glucocorticoids (synthesized in the neighboring adrenal cortex).

other important outcomes such as successful life transition (Lavallee, 2005), the quality of sport experiences (Lindsay, Maynard, & Thomas, 2005; Newburg et al, 2002), life skills (Danish & Nellen, 1997), and sociomoral growth of children (S C Miller, Bredemeier, & Shields, 1997) Model The second layer in the mental training process, emanating from philosophy, is one s model of intervention, or the overarching thematic framework from which specific mental training strategies and techniques are developed and utilized The mental skills training literature abounds with the description of many models of intervention These include systems models for team, organizational, and family interventions (Hellstedt, 1995; Zimmerman, Protinsky, & Zimmerman, 1994), self-regulatory or cognitive-behavioral models (Boutcher & Rotella, 1987; Hanin, 2000; Kirschenbaum & Wittrock, 1984; Moore & Stevenson, 1994; Singer, 1988), behavioral management models (G L Martin & Toogood, 1997; S B.

By contrast, the conversion of NE to E, which requires the functional expression of phenylethanolamine-N -methyltransferase (PNMT), the nal enzyme in the catecholamine synthesizing cascade, might require induction by glucocorticoids, such as dexamethasone [Doupe et al, 1985; Anderson and Michelson, 1989; Finotto et al, 1999; Wong, 2003] The details of the chromaf nergic differentiation process, as fascinating as they may be, are beyond the scope of this article and can be found in a recent review [Huber et al, 2002] Suf ce it to say that, regardless of the effect of glucocorticoids, homotypic and heterotypic 3-D cell-cell interactions during organogenesis appear to be pivotal for the organotypic differentiation of the parenchymal cells in the adrenal medulla [Lelkes and Unsworth, 1992].

Chromaf n cells have been used for many decades as an easily available model system for studying mechanisms of neurotransmitter synthesis and release, speci cally of cholinergic neuronal or neuroendocrine stimulus-secretion-synthesis coupling Indeed, a recent PubMed search for chromaf n cells (August 2005) revealed some 3900 relevant hits In contrast to bona de chromaf n cells, PC12 pheochromocytoma cells are predominantly dopaminergic, that is, they express tyrosine hydroxylase (TH) and dopamine- -hydroxylase (DBH), and synthesize and secrete small amounts of NE [Greene and Tischler, 1976] In the hands of most (but not all) investigators, PC12 cells do not spontaneously express PNMT or epinephrine, thus suggesting that these cells originated from the NE phenotype of chromaf n cells In the past 25 years PC12 cells have become an enormously popular model, mainly for studying fundamental mechanisms of neuronal differentiation and mechanisms of neurotransmitter synthesis and release.